Merge pull request #184 from ake123/master

correction to bib file
openresearchlabs · Jan 20, 2025 · f3ebfb5 · f3ebfb5
2 parents 86476f8 + 67a976a
commit f3ebfb5
Showing 1 changed file with 0 additions and 62 deletions.
diff --git a/content/publication_resources/bibtex/lahti.bib b/content/publication_resources/bibtex/lahti.bib
@@ -837,68 +837,6 @@ @Article{Palmu2022
   keywords =	 {bioscience}
 }
 
-
-@article {Husso2022,
-  author =	 {Husso, Aleksi and Pessa-Morikawa, Tiina and
-                  Koistinen, Ville Mikael and Karkkainen, Olli and
-                  Lahti, Leo and Iivanainen, Antti and Hanhineva, Kati
-                  and Niku, Mikael},
-  title =	 {Impacts of maternal microbiota and microbial
-                  metabolites on fetal intestine, brain and placenta},
-  year =	 2022,
-  doi =		 {10.1101/2022.07.01.498433},
-  publisher =	 {Cold Spring Harbor Laboratory},
-  abstract =	 {The maternal microbiota modulates fetal development,
-                  but the mechanisms of these earliest host-microbe
-                  interactions are unclear. We compared full-term
-                  fetuses from germ-free (GF) and normally colonized
-                  mouse dams by gene expression profiling and
-                  non-targeted metabolomics. The developing immune
-                  system was strongly dependent on the maternal
-                  microbial status. In the fetal intestine, critical
-                  components mediating host-microbe interactions were
-                  differentially expressed. In fetal brain and
-                  placenta, interferon and inflammatory signaling were
-                  downregulated in germ-free fetuses. Neural system
-                  development and function, translation and RNA
-                  metabolism, and regulation of energy metabolism were
-                  significantly affected at the gene expression
-                  level. These impacts were strongly associated with
-                  microbial metabolite concentrations in the fetal
-                  tissues, suggesting that they are largely, although
-                  perhaps not exclusively mediated by maternal
-                  microbial metabolites absorbed through
-                  placenta. Several aryl sulfates were among the
-                  compounds strongly associated with gene expression
-                  differences. The germ-free fetus may suffer from
-                  depletion of queuine, a bacterial hypermodified
-                  nucleobase essential for eukaryotic tRNA stability
-                  and function.Competing Interest StatementThe authors
-                  have declared no competing interest.},
-  URL =
-                  {https://www.biorxiv.org/content/early/2022/07/03/2022.07.01.498433},
-  eprint =
-                  {https://www.biorxiv.org/content/early/2022/07/03/2022.07.01.498433.full.pdf},
-  keywords =	 {bioscience},
-  journal =	 {bioRxiv}
-}
-
-@Article{Liu2022,
-  author =	 {Yang Liu and Shu Mei Teo and Guillaume Meric and
-                  Howard HF Tang and Qiyun Zhu and Jon G Sanders and
-                  Yoshiki Vazquez-Baeza and Karin Verspoor and Ville A
-                  Vartiainen and Pekka Jousilahti and Leo Lahti and
-                  Teemu Niiranen and Aki S Havulinna and Rob Knight
-                  and Veikko Salomaa and Michael Inouye},
-  title =	 {The gut microbiome is a significant risk factor for
-                  future chronic lung disease},
-  journal =	 {{medRxiv}},
-  year =	 {2022},
-  month =	 03,
-  doi =		 {10.1101/2022.03.22.22272736},
-  keywords =	 {bioscience}
-}
-
 @Article{Khalighi2022,
   author =	 {Moein Khalighi and Guilhem Sommeria-Klein and Didier
                   Gonze and Karoline Faust and Leo Lahti},